Annual Meeting of the NCI Cohort Consortium (Abstract Submission): Submission #10
Submission information
Submission Number: 10
Submission ID: 150783
Submission UUID: f131f9a1-dda3-478d-8e35-d8cc2eb94194
Submission URI: /egrp/cohortconsortium/abstracts?cid=eb_govdel
Submission Update: /egrp/cohortconsortium/abstracts?cid=eb_govdel&token=3CRzr0MpWSATdtAjoE5zgPpiJItgXI0CQ-lNiqwPB1M
Created: Wed, 09/03/2025 - 13:13
Completed: Wed, 09/03/2025 - 13:42
Changed: Wed, 09/03/2025 - 13:42
Remote IP address: 10.208.24.230
Submitted by: Anonymous
Language: English
Is draft: No
Webform: Cohort 2025 (Abstracts Submission)
Presenter's First Name: | Ting |
---|---|
Presenter's Last Name: | Zhang |
Title (eg: professor, assistant professor, chair, etc): | postdoctoral fellow |
Degree(s) | PhD |
Contact Email: | ting.zhang3@nih.gov |
Organization: | National Cancer Institute |
Project Title: | Blood proteomic profiles and pancreatic ductal adenocarcinoma risk – A Mendelian randomization and observational analysis |
Additional Authors |
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Abstract: | Background: Proteomic biomarkers hold promises for early detection and etiologic insights into pancreatic ductal adenocarcinoma (PDAC). We investigated associations between genetically predicted plasma proteins and PDAC risk using Mendelian randomization (MR) and assessed identified proteins with incident PDAC in two prospective cohorts. Methods: We conducted proteome-wide MR by applying cis-acting protein quantitative trait loci (pQTLs, P<5×10-8) identified from genome-wide association studies of plasma proteins in the UK Biobank (UKB, n=34,557; 2923 proteins) and deCODE (n=35,559; 4719 proteins), respectively, to summary statistics from a PDAC GWAS meta-analysis of PanScan1–3/PanC4/FinnGen (10,244 cases, 360,535 controls of European ancestry). We validated the top MR-identified proteomic-PDAC associations using Cox proportional hazards models within UKB (n=52,551, 133 cases) and Atherosclerosis Risk in Communities (ARIC) study (n=9476; 88 cases) cohorts and combined using fixed-effect meta-analysis. Results: The proteome-wide MR identified 44 proteins significantly associated with PDAC risk after Bonferroni correction (PUKB<2.7×10-5 or PdeCODE <3.0×10-5), of which 17 were cross-validated in both datasets (P<0.05). Of the 44, eight proteins were previously identified, including ABO, CTRB1, B4GALT1, REG1A, FUT3, AMY2A, AMY2B, and NCF1 (OR=0.80–1.27 per standard deviation increment). The remaining 36 proteins were newly identified. In prospective analyses, higher plasma ABO and ITIH3 were associated with incident PDAC in UKB-ARIC meta-analysis (HR=1.19 and 1.20 respectively). Conclusion/Discussion: We identified multiple genetically predicted plasma proteins associated with PDAC risk, among which associations for measured plasma ABO and ITIH3 were further validated using in two prospective cohorts. These proteins highlight promising candidates for future investigations as PDAC prevention targets. |